Mechanisms of Absence Seizures
DOI:
https://doi.org/10.15844/pedneurbriefs-9-3-4Keywords:
G-Aminobutyric Acid, Absence Seizures, Synchronous SpikeAbstract
A unifying hypothesis for the pathogenesis of absence seizures, involving the thalamocortical circuitry, is proposed in a neurological progress report from the University of Southern California School of Medicine, Childrens Hospital Los Angeles.Published
1995-03-01
Issue
Section
Seizure Disorders
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Copyright (c) 1995 The Author(s)
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