1043-3155 Pediatr Neurol Briefs pedneurbriefs Pediatr Neurol Briefs Pediatric Neurology Briefs Pediatr Neurol Briefs 2166-6482 1043-3155 2166-3155 Pediatric Neurology Briefs Publishers Chicago, IL, USA PNB-6-42-a 10.15844/pedneurbriefs-6-6-2 Neonatal Encephalopathy Neurology Pediatrics Nervous System Diseases Child Development Brain Diseases Neurosurgery Child Infant Birth Asphyxia, Cerebral Metabolism and Head Size http://orcid.org/0000-0002-0173-7931 Millichap J. Gordon MD 1 2 * Division of Neurology, Children's Memorial Hospital, Chicago, IL Departments of Pediatrics and Neurology, Northwestern University Feinberg School of Medicine, Chicago, IL Correspondence: Dr. J. Gordon Millichap, E-mail: jgmillichap@northwestern.edu 06 1992 01 07 2016 6 6 42 42 Copyright: © 1992 The Author(s) 1992 This work is licensed under the Creative Commons Attribution 4.0 International License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Relation between cerebral oxidative metabolism following birth asphyxia, and neurodevelopmental outcome and brain growth at one year

Studies of cerebral oxidative metabolism were carried out by phosphorus magnetic resonance spectroscopy during the first week of life in 52 infants with birth asphyxia admitted to the Neonatal Unit at University College Hospital, London.

 Birth Asphyxia Head Growth Hypernatremia

Studies of cerebral oxidative metabolism were carried out by phosphorus magnetic resonance spectroscopy during the first week of life in 52 infants with birth asphyxia admitted to the Neonatal Unit at University College Hospital, London. Cerebral phosphocreatine/inorganic phosphate concentration ratio was used as an index of oxidative metabolism and correlated with neurodevelopmental outcome and head growth at 1 year. [1]

COMMENT. The use of these neuroimaging and biochemical techniques should help in the prediction of outcome following neonatal encephalopathy. However, the complexity of the techniques may detract from their value in practice. [2]

Central diabetes insipidus as an unusual complication of hypoxic brain damage is described in 2 children at the Department of Pediatrics, Juntendo University School of Medicine, Tokyo, Japan [3]. Both patients developed cardiopulmonary arrest after choking and both had hypernatremia and low urinary antidiuretic hormone concentrations in the terminal stages. The most common causes of central diabetes insipidus are tumors or trauma in the neurohypophyseal area.

 Roth SC Edwards AD Cady EB Delpy DT Wyatt JS Azzopardi D Relation between cerebral oxidative metabolism following birth asphyxia, and neurodevelopmental outcome and brain growth at one year Dev Med Child Neurol 1992 Apr 34 4 285 295 10.1111/j.1469-8749.1992.tb11432.x 1572514 Bax M Birth asphyxia Dev Med Child Neurol 1992 Apr 34 4 283 284 Editorial 10.1111/j.1469-8749.1992.tb11431.x 1572513 Arisaka O Arisaka M Ikebe A Niijima A Shimura N Hosaka A Central diabetes insipidus in hypoxic brain damage Child’s Nerv Syst 1992 Mar 8 2 81 82 10.1007/BF00298445 1591751